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human lung epithelial cell line beas 2b  (ATCC)


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    ATCC human lung epithelial cell line beas 2b
    Human Lung Epithelial Cell Line Beas 2b, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 3986 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/lung+epithelial+cell+line+beas+2b/BEAS-2B/pm42128262-50-10-23
    Average 99 stars, based on 3986 article reviews
    human lung epithelial cell line beas 2b - by Bioz Stars, 2026-09
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    Article Title: Local anesthetic ropivacaine inhibits non-small cell lung cancer progression by modulating the hsa_circ_0001320/miR-518a-5p axis
    Article Snippet: Human NSCLC cell lines (H1299, H1975, and A549) and the normal lung epithelial cell line BEAS-2B were purchased from American Type Culture Collection (ATCC; USA).

    Article Title: Targeting SIRT3 signaling alleviates lung carcinoma progression by reducing hyperinsulinemia in postmenopausal obese mice: Protective intervention of betaxanthin
    Article Snippet: The lung epithelial cell line BEAS-2B (#CRL-3588) and MLg (#CCL-206) also were purchased from American Type Culture Collection (ATCC, Manassas, VA).

    Article Title: PD-L1 knockdown suppresses vasculogenic mimicry of non-small cell lung cancer by modulating ZEB1-triggered EMT
    Article Snippet: The human non-small cell lung cancer (NSCLC) cell lines A549, H1299, and PC9, as well as the normal lung epithelial cell line BEAS-2B, were acquired from the American Type Culture Collection (ATCC).

    Article Title: N4-acetylcytidine modification of LINC02802 promotes non-small cell lung cancer progression by modulating mitochondrial NAD+/NADH ratio
    Article Snippet: This study included the normal lung epithelial cell line BEAS-2B (human bronchial epithelial cells) as well as NSCLC cell lines from ATCC.

    Article Title: DH_27, a right-half derivative of renieramycin T, induce apoptosis through inhibition of mTOR signaling for lung cancer suppression.
    Article Snippet: NSCLC cell lines (H460 and H23) and lung epithelial cell line (BEAS-2B) were sourced from the American Type Culture Collection (ATCC; Manassas, VA, USA), whereas an immortalized dermal papilla cells (DP) were acquired from Applied Biological Materials Inc. (Richmond, BC, Canada).

    Article Title: Controlling glycolysis to generate characteristic volatile organic compounds of lung cancer cells
    Article Snippet: The lung cancer cell lines A549, PC-9, NCI-H460, and the normal lung epithelial cell line BEAS-2B used in this experiment were purchased from the American Type Culture Collection (ATCC) and cryopreserved in our laboratory’s liquid nitrogen tank at a temperature of − 192 °C.

    Article Title: Controlling glycolysis to generate characteristic volatile organic compounds of lung cancer cells.
    Article Snippet: The lung cancer cell lines A549, PC-9, NCI-H460, and the normal lung epithelial cell line BEAS-2B used in this experiment were purchased from the American Type Culture Collection (ATCC) and cryopreserved in our laboratory’s liquid nitrogen tank at a temperature of − 192 °C.

    Cell Culture:

    Article Title: Transcription Factor FOSL1 Promotes Cisplatin Resistance in Non-Small Cell Lung Cancer Cells by Modulating the Wnt3a/β-Catenin Signaling through Upregulation of PLIN3 Expression
    Article Snippet: .. The NSCLC cell lines H2030 (ATCC), H2405, H1755, H2347, H1703, and the lung epithelial cell line BEAS-2B were sourced from American Type Culture Collection (ATCC, Manassas, MD, USA) and cultured in a humidified incubator at 37 °Cwith 5%CO2. .. Cells were maintained in RPMI-1640 medium (Sigma-Aldrich, Shanghai, China) supplemented with 10% fetal bovine serum (FBS, Sigma-Aldrich, Shanghai, China).



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    THOC3 expression is upregulated in LC and is associated with unfavorable prognoses. (A, B) The mRNA expression of THOC3 in LUAD and LUSC tissues, as well as normal lung tissues, was obtained from the GEPIA database. (C) Real-time PCR analysis of THOC3 mRNA expression in cancerous and adjacent non-cancerous tissues from LC patients. (D) Western blot analysis of THOC3 protein levels in cancerous and adjacent tissues from LC patients. (E) Real-time PCR analysis of THOC3 mRNA expression in LC cell lines (H1975, A549, H1299, and PC9) and the normal bronchial epithelial cell <t>line</t> <t>BEAS-2B</t> (F) Western blot analysis of THOC3 protein expression in LC cell lines (H1975, A549, H1299, and PC9) and BEAS-2B cells. (G) Real-time PCR analysis of THOC3 mRNA expression in normal lung tissues and LC samples from different clinical grades. (H) Western blot analysis of THOC3 protein levels in normal lung tissues and LC samples from various clinical stages. (I, J, K) Kaplan-Meier survival curves illustrating the overall survival (OS) of LC patients with high or low THOC3 expression from the TCGA database. (L) Kaplan-Meier survival curves depicting disease-free survival (DFS) in high vs. low THOC3-expressing LC patients from the TCGA database. Each experiment was performed in triplicate. Data are expressed as mean ± SEM; * P < 0.05.
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    human lung epithelial beas 2b cell line  (ATCC)
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    ATCC human lung epithelial beas 2b cell line
    THOC3 expression is upregulated in LC and is associated with unfavorable prognoses. (A, B) The mRNA expression of THOC3 in LUAD and LUSC tissues, as well as normal lung tissues, was obtained from the GEPIA database. (C) Real-time PCR analysis of THOC3 mRNA expression in cancerous and adjacent non-cancerous tissues from LC patients. (D) Western blot analysis of THOC3 protein levels in cancerous and adjacent tissues from LC patients. (E) Real-time PCR analysis of THOC3 mRNA expression in LC cell lines (H1975, A549, H1299, and PC9) and the normal bronchial epithelial cell <t>line</t> <t>BEAS-2B</t> (F) Western blot analysis of THOC3 protein expression in LC cell lines (H1975, A549, H1299, and PC9) and BEAS-2B cells. (G) Real-time PCR analysis of THOC3 mRNA expression in normal lung tissues and LC samples from different clinical grades. (H) Western blot analysis of THOC3 protein levels in normal lung tissues and LC samples from various clinical stages. (I, J, K) Kaplan-Meier survival curves illustrating the overall survival (OS) of LC patients with high or low THOC3 expression from the TCGA database. (L) Kaplan-Meier survival curves depicting disease-free survival (DFS) in high vs. low THOC3-expressing LC patients from the TCGA database. Each experiment was performed in triplicate. Data are expressed as mean ± SEM; * P < 0.05.
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    THOC3 expression is upregulated in LC and is associated with unfavorable prognoses. (A, B) The mRNA expression of THOC3 in LUAD and LUSC tissues, as well as normal lung tissues, was obtained from the GEPIA database. (C) Real-time PCR analysis of THOC3 mRNA expression in cancerous and adjacent non-cancerous tissues from LC patients. (D) Western blot analysis of THOC3 protein levels in cancerous and adjacent tissues from LC patients. (E) Real-time PCR analysis of THOC3 mRNA expression in LC cell lines (H1975, A549, H1299, and PC9) and the normal bronchial epithelial cell line BEAS-2B (F) Western blot analysis of THOC3 protein expression in LC cell lines (H1975, A549, H1299, and PC9) and BEAS-2B cells. (G) Real-time PCR analysis of THOC3 mRNA expression in normal lung tissues and LC samples from different clinical grades. (H) Western blot analysis of THOC3 protein levels in normal lung tissues and LC samples from various clinical stages. (I, J, K) Kaplan-Meier survival curves illustrating the overall survival (OS) of LC patients with high or low THOC3 expression from the TCGA database. (L) Kaplan-Meier survival curves depicting disease-free survival (DFS) in high vs. low THOC3-expressing LC patients from the TCGA database. Each experiment was performed in triplicate. Data are expressed as mean ± SEM; * P < 0.05.

    Journal: Translational Oncology

    Article Title: THOC3 interacts with epithelial-to-mesenchymal transition to promote non-small cell lung cancer carcinoma progression through STAT3 signaling pathway

    doi: 10.1016/j.tranon.2026.102741

    Figure Lengend Snippet: THOC3 expression is upregulated in LC and is associated with unfavorable prognoses. (A, B) The mRNA expression of THOC3 in LUAD and LUSC tissues, as well as normal lung tissues, was obtained from the GEPIA database. (C) Real-time PCR analysis of THOC3 mRNA expression in cancerous and adjacent non-cancerous tissues from LC patients. (D) Western blot analysis of THOC3 protein levels in cancerous and adjacent tissues from LC patients. (E) Real-time PCR analysis of THOC3 mRNA expression in LC cell lines (H1975, A549, H1299, and PC9) and the normal bronchial epithelial cell line BEAS-2B (F) Western blot analysis of THOC3 protein expression in LC cell lines (H1975, A549, H1299, and PC9) and BEAS-2B cells. (G) Real-time PCR analysis of THOC3 mRNA expression in normal lung tissues and LC samples from different clinical grades. (H) Western blot analysis of THOC3 protein levels in normal lung tissues and LC samples from various clinical stages. (I, J, K) Kaplan-Meier survival curves illustrating the overall survival (OS) of LC patients with high or low THOC3 expression from the TCGA database. (L) Kaplan-Meier survival curves depicting disease-free survival (DFS) in high vs. low THOC3-expressing LC patients from the TCGA database. Each experiment was performed in triplicate. Data are expressed as mean ± SEM; * P < 0.05.

    Article Snippet: The human normal lung epithelial cell line BEAS-2B was obtained from the American Type Culture Collection.

    Techniques: Expressing, Real-time Polymerase Chain Reaction, Western Blot